<1>. 1.5 Tesla MRI of brain on 2009.04.02 with T1 weighted echo
 train spin echo, T2 weighted echo train spin echo, T2 weighted
 fluid-attenuated inversion recovery echo train spin echo,
 diffusion-weighted echo-planar MR imaging, single voxel proton
 MR spectroscopy, and with intravenous gadolinium-enhanced images:
<2>. 1.5 Tesla MRI of brain on 2009.04.03 for further evaluation with
 T2 weighted fluid-attenuated inversion recovery echo train spin
 echo with multiplanar reconstruction and 3D volume rendering,
 diffusion-weighted echo-planar MR imaging with apparent diffusion
 coefficient maps, fractional anisotropy maps, and colored-fractional
 anisotropy maps, and without intravenous contrast:
<3>. 1.5 Tesla MRI of spine with T1 weighted echo train spin echo,
 T2 weighted echo train spin echo, and without intravenous contrast:

1. Numerous long T2 lesions (fig 1,2,3,and 6) without significant
   contrast enhancement or restricted water diffusibility (fig 4),
   around 0.5-1.5cm, in central nervous system, chiefly in
   subcortical white matter of bilateral cerebral hemispheres,
   but also including pons, cerebellum, and along spinal cord
   (fig 5).
2. Presence of mild mass effect of above lesions, predominant over
   spinal cord.
3. From above findings, acute disseminated encephalomyelitis (ADEM)
   may be considered firstly.
   Less possibility: multiple sclerosis, Langerhans cell histiocytosis,
   subacute sclerosing panencephalitis, progressive multifocal
   leukoencephalopathy, etc.